3 MAIN  MECHANISMS  IN WHICH FENBENDAZOLE KILLS CANCER.

1) Apoptosis induction. Cancer cells are stopped from dividing by inhibiting cell division (mitosis) and by inducing programmed cell death (apoptosis), all of which result in tumor eradication.

2) Inhibition of glucose uptake in cancer cells. Malignant cells are known to have an enormous glucose uptake. Cancer cells normally consume glucose 200 times faster than ordinary cells. This can be seen in PET scans – the metabolically active sites, which use more radioactive glucose can be clearly seen. Fenbendazole limits cancer cell fueling with sugar by limiting the glucose uptake, while decreasing the amount of canals that take glucose into the cancer cells from the blood.

3) Reactivation of the p53 gene. There is an increasing number of studies that confirm the fact that fenbendazole might truly increase the strongest tumor suppressor in our bodies – p53.
“(These) various modes of action mean that there is almost no possibility for cancer cells to develop resistance to the drug. Because it targets several routes, if resistance develops to one mechanism of action, fenbendazole can still affect cancer cells in other ways. Furthermore, fenbendazole may be used with other anticancer treatments owing to its unique mechanism of action and low toxicity.”